Consider two random vectors $\wt{\bx} \in \R^p$ and $\wt{\by} \in \mathbb R^q$ of the forms $\wt{\bx}=A \mathbf z + \bC_1^{1/2}\mathbf x $ and $\wt{\by}=B \mathbf z + \bC_2^{1/2}\mathbf y $, where $\mathbf x \in \R^p$, $\mathbf y \in \R^q$ and $\mathbf z\in \R^r$ are independent random vectors with i.i.d. entries of zero mean and unit variance, $\bC_1$ and $\bC_2$ are $p \times p$ and $q\times q$ deterministic population covariance matrices, and $A$ and $B$ are $p \times r$ and $q\times r$ deterministic factor loading matrices. With $n$ independent observations of $(\wt{\mathbf x},\wt{\mathbf y})$, we study the sample canonical correlations between $\wt{\bx} $ and $\wt{\by}$. We consider the high-dimensional setting with finite rank correlations, that is, ${p}/{n}\to c_1$ and ${q}/{n}\to c_2$ as $n\to \infty$ for some constants $c_1\in (0,1)$ and $c_2\in (0,1-c_1)$, and $r$ is a fixed integer. Let $t_1\ge t_2 \ge \cdots\ge t_r\ge 0$ be the squares of the nontrivial population canonical correlation coefficients between $\wt {\bx}$ and $\wt{\by}$, and let $\wt\lambda_1 \ge \wt\lambda_2\ge \cdots \ge \wt\lambda_{p\wedge q}\ge 0$ be the squares of the sample canonical correlation coefficients. If the entries of $\mathbf x$, $\mathbf y$ and $\mathbf z$ are i.i.d. Gaussian, then the following dichotomy has been shown in \cite{CCA} for a fixed threshold $t_c \in(0, 1)$: for any $1\le i \le r$, if $t_i < t_c$, then $\wt\lambda_i$ converges to the right-edge $\lambda_+$ of the limiting eigenvalue spectrum of the sample canonical correlation matrix, and moreover, $n^{2/3}(\wt\lambda_i-\lambda_+)$ converges weakly to the Tracy-Widom law; if $t_i>t_c$, then $\wt\lambda_i$ converges to a deterministic limit $\theta_i \in (\lambda_+, 1)$ that is determined by $c_1$, $c_2$ and $t_i$. In this paper, we prove that these results hold universally under the sharp fourth moment conditions on the entries of $\mathbf x$, $\mathbf y$ and $\mathbf z$. Moreover, we prove the results in full generality, in the sense that they also hold for near-degenerate $t_i$'s and for $t_i$'s that are close to the threshold $t_c$. Finally, we also provide almost sharp convergence rates for the sample canonical correlation coefficients under a general $a$-th moment assumption.

We consider Green's functions $G(z):=(H-z)^{-1}$ of Hermitian random band matrices $H$ on the $d$-dimensional lattice $(\Z/L\Z)^d$. The entries $h_{xy}=\overline h_{yx}$ of $H$ are independent centered complex Gaussian random variables with variances $s_{xy}=\mathbb E|h_{xy}|^2$, which satisfy a banded profile so that $s_{xy}$ is negligible if $|x-y|$ exceeds the band width $W$. For any fixed $n\in \N$, we construct an expansion of the $T$-variable, $T_{xy}=|m|^2 \sum_{\alpha}s_{x\alpha}|G_{\alpha y}|^2$, with an error $\OO(W^{-nd/2})$, and use it to prove a local law on the Green's function. This $T$-expansion was the main tool to prove the delocalization and quantum diffusion of random band matrices for dimensions $d\ge 8$ in part I \cite{PartI_high} of this series.

We consider Hermitian random band matrices $H=(h_{xy})$ on the $d$-dimensional lattice $(\Z/L\Z)^d$. The entries $h_{xy}$ are independent (up to Hermitian conditions) centered complex Gaussian random variables with variances $s_{xy}=\mathbb E|h_{xy}|^2$. The variance matrix $S=(s_{xy})$ has a banded structure so that $s_{xy}$ is negligible if $|x-y|$ exceeds the band width $W$. In dimensions $d\ge 8$, we prove that, as long as $W\ge L^\e$ for a small constant $\e>0$, with high probability most bulk eigenvectors of $H$ are delocalized in the sense that their localization lengths are comparable to $L$. Denote by $G(z)=(H-z)^{-1}$ the Green's function of $H$. For $\im z\gg W^2/L^2$, we also prove a widely used criterion in physics for quantum diffusion of this model, namely, the leading term in the Fourier transform of $\E|G_{xy}(z)|^2$ with respect to $x-y$ is of the form $(\im z + a(p))^{-1}$ for some $a(p)$ quadratic in $p$, where $p$ is the Fourier variable. Our method is based on an expansion of $T_{xy}=|m|^2 \sum_{\alpha}s_{x\alpha}|G_{\alpha y}|^2$ and it requires a self-energy renormalization up to error $W^{-K}$ for any large constant $K$ independent of $W$ and $L$. We expect that this method can be extended to non-Gaussian band matrices.

We propose a deep learning-based method for object detection in UAV-borne thermal images that have the capability of observing scenes in both day and night. Compared with visible images, thermal images have lower requirements for illumination conditions, but they typically have blurred edges and low contrast. Using a boundary-aware salient object detection network, we extract the saliency maps of the thermal images to improve the distinguishability. Thermal images are augmented with the corresponding saliency maps through channel replacement and pixel-level weighted fusion methods. Considering the limited computing power of UAV platforms, a lightweight combinational neural network ComNet is used as the core object detection method. The YOLOv3 model trained on the original images is used as a benchmark and compared with the proposed method. In the experiments, we analyze the detection performances of the ComNet models with different image fusion schemes. The experimental results show that the average precisions (APs) for pedestrian and vehicle detection have been improved by 2%~5% compared with the benchmark without saliency map fusion and MobileNetv2. The detection speed is increased by over 50%, while the model size is reduced by 58%. The results demonstrate that the proposed method provides a compromise model, which has application potential in UAV-borne detection tasks.

The stochasticity of gene expression is manifested in the fluctuations of mRNA and protein copy numbers within a cell lineage over time. While data of this type can be obtained for many generations, most mathematical models are unsuitable to interpret such data since they assume non-growing cells. Here we develop a theoretical approach that quantitatively links the frequency content of lineage data to subcellular dynamics. We elucidate how the position, height, and width of the peaks in the power spectrum provide a distinctive fingerprint that encodes a wealth of information about mechanisms controlling transcription, translation, replication, degradation, bursting, promoter switching, cell cycle duration, cell division, gene dosage compensation, and cell size homeostasis. Predictions are confirmed by analysis of single-cell Escherichia coli data obtained using fluorescence microscopy. Furthermore, by matching the experimental and theoretical power spectra, we infer the temperature-dependent gene expression parameters, without the need of measurements relating fluorescence intensities to molecule numbers.