We use Coulomb branch indices of Argyres-Douglas theories on S1×L(k,1) to quantize moduli spaces H of wild/irregular Hitchin systems. In particular, we obtain formulae for the "wild Hitchin characters" -- the graded dimensions of the Hilbert spaces from quantization -- for four infinite families of H, giving access to many interesting geometric and topological data of these moduli spaces. We observe that the wild Hitchin characters can always be written as a sum over fixed points in H under the U(1) Hitchin action, and a limit of them can be identified with matrix elements of the modular transform STkS in certain two-dimensional chiral algebras. Although naturally fitting into the geometric Langlands program, the appearance of chiral algebras, which was known previously to be associated with Schur operators but not Coulomb branch operators, is somewhat surprising.

This paper deals with the derivation and analysis of the the Hall Magneto-Hydrodynamic equations. We first provide a derivation of this system from a two-fluids Euler-Maxwell system for electrons and ions, through a set of scaling limits. We also propose a kinetic formulation for the Hall-MHD equations which contains as fluid closure different variants of the Hall-MHD model. Then, we prove the existence of global weak solutions for the incompressible viscous resistive Hall-MHD model. We use the particular structure of the Hall term which has zero contribution to the energy identity. Finally, we discuss particular solutions in the form of axisymmetric purely swirling magnetic fields and propose some regularization of the Hall equation.

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Using a geometric method, we characterize all entire functions that transform the Bloch space into a Bergman space by superposition in terms of their order and type. We also prove that all superposition operators induced by such entire functions act boundedly. Similar results hold for superpositions from BMOA into Bergman spaces and from the Bloch space into certain weighted Hardy spaces.

Over time, a population acquires neutral genetic substitutions as a consequence of random drift. A famous result in population genetics asserts that the rate, K, at which these substitutions accumulate in the population coincides with the mutation rate, u, at which they arise in individuals: K = u. This identity enables genetic sequence data to be used as a “molecular clock” to estimate the timing of evolutionary events. While the molecular clock is known to be perturbed by selection, it is thought that K = u holds very generally for neutral evolution. Here we show that asymmetric spatial population structure can alter the molecular clock rate for neutral mutations, leading to either K<u or K>u. Our results apply to a general class of haploid, asexually reproducing, spatially structured populations. Deviations from K = u occur because mutations arise unequally at different sites and have different probabilities of fixation depending on where they arise. If birth rates are uniform across sites, then K
u. In general, K can take any value between 0 and Nu. Our model can be applied to a variety of population structures. In one example, we investigate the accumulation of genetic mutations in the small intestine. In another application, we analyze over 900 Twitter networks to study the effect of network topology on the fixation of neutral innovations in social evolution.